Energy systems
How muscles remake ATP: the phosphagen, glycolytic and oxidative systems, how much each supplies at different effort durations, which fuel they burn, what lactate does, how to train each system, and how to keep energy up day to day. Running sessions are in endurance, lifting in strength training, and daily eating in nutrition and hydration. For work, power and energy as physics, see physics fundamentals.
ATP: the energy currency
Muscle contraction, calcium pumping and ion pumps all run on ATP splitting to ADP and phosphate. The stored ATP is tiny, so it has to be remade as fast as it is used.
| Fact | Value | Source |
|---|---|---|
| ATP stored in muscle | ≈ 5 mmol/kg wet muscle | Hargreaves & Spriet 2020 |
| ATP use, all-out sprint (≈ 900 W) | 3.7 mmol/kg/s: stored ATP alone would last under 2 s | Hargreaves & Spriet 2020 |
| ATP use, ≈ 75% VO₂max (≈ 200 W) | 0.4 mmol/kg/s: stored ATP would last ≈ 15 s | Hargreaves & Spriet 2020 |
| ATP level in hard exercise | fairly well defended: ≈ 20% lower in very intense work, 30% lower after a 30 s sprint | Hargreaves & Spriet 2020; Bogdanis et al. 1995 |
Ways to remake ATP
| Pathway | Reaction (simplified) | ATP gained |
|---|---|---|
| phosphagen | PCr + ADP → ATP + creatine | 1 per PCr |
| glycolysis to lactate | glycogen (1 glucose unit) → 2 lactate | 3 |
| glucose oxidation | glucose + 6 O₂ → 6 CO₂ + 6 H₂O | ≈ 36 by the traditional count; lower with modern estimates |
| fat oxidation | palmitate + 23 O₂ → 16 CO₂ + 16 H₂O | ≈ 130 by the traditional count; also lower |
Yields from Hargreaves & Spriet 2020 (Box 1). The ratios between fuels hold whichever count you use.
The three systems
All three run all the time. What changes with intensity and duration is how much each one supplies. Fast systems have high rate (power) but small capacity (total ATP); slow systems are the reverse.
| Phosphagen (ATP–PCr) | Glycolytic ("anaerobic", fast glycolysis) | Oxidative (aerobic) | |
|---|---|---|---|
| where | cytosol | cytosol | mitochondria |
| fuel | phosphocreatine (PCr) | muscle glycogen, blood glucose | carbohydrate, fat (a little protein) |
| needs O₂? | no | no | yes |
| rate (power) | highest | high | lowest; carbohydrate faster than fat |
| capacity | smallest: PCr ≈ 75 mmol/kg dry muscle, mostly used in 10–15 s all-out | ≈ 3× PCr: ≈ 225 mmol/kg dry muscle over 30–90 s | very large: glycogen, then fat |
| main role | first ≈ 10 s of maximal work; jumps, throws, lifts, sprints | 10 s to ≈ 1–2 min of hard work | dominant beyond ≈ 75–80 s; everything submaximal |
| by-products | creatine, phosphate (Pi) | lactate, H⁺ | CO₂, water |
| what limits it | PCr runs out | rising acidity, not lack of glycogen | O₂ delivery and mitochondria; glycogen in long events |
| recovery | PCr half back in ≈ 20–60 s; near full in several minutes | as PCr recovers and acidity clears | glycogen: ≈ 24 h to refill with enough carbohydrate |
Sources: capacities, limits and timing from Hargreaves & Spriet 2020; crossover time from Gastin & Suppiah 2026; glycogen refill from Murray & Rosenbloom 2018; PCr recovery below.
PCr resynthesis
| After | PCr recovered | Source |
|---|---|---|
| exhaustive dynamic exercise | biphasic: fast half-time 21–22 s, slow half-time over 170 s | Harris et al. 1976 |
| a 30 s all-out cycle sprint | 65% at 1.5 min, 85.5% at 6 min; half-time ≈ 57 s; power recovers in step with PCr | Bogdanis et al. 1995 |
| with blood flow blocked (cuff) | none; resynthesis needs oxygen | Harris et al. 1976 |
So a 6–10 s sprint needs 1.5–3 min rest to repeat near full power; after a 30 s effort, even 6 min is not quite enough. PCr is remade aerobically, so a better aerobic base speeds recovery between sprints and sets.
Who does what in a short sprint
| Effort | Energy split | Source |
|---|---|---|
| first 6 s cycle sprint | PCr and anaerobic glycolysis about equal; PCr fell 57% | Gaitanos et al. 1993 |
| 30 s Wingate test | 18.6% aerobic, 31.1% phosphagen, 50.3% glycolytic | Beneke et al. 2002 |
| last 5 s of a 30 s sprint | ≈ 50% aerobic; VO₂ reaches 70–100% of VO₂max | Hargreaves & Spriet 2020 |
The oxidative system runs in the mitochondria: the Krebs cycle in the matrix, the electron transport chain and ATP synthase on the folded inner membrane (cristae). Endurance training adds mitochondria.
Image: Mariana Ruiz Villarreal (LadyofHats), public domain, via Wikimedia Commons (opens in a new tab).
Watch for how the three systems overlap rather than switch on in turn, and which fuel each uses.
Contribution by effort duration
For a single all-out effort, the aerobic share rises fast with duration. Gastin & Suppiah's 2026 systematic review (102 studies, 311 data points) put the 50:50 point at 78.6 s (≈ 75–80 s). Its estimates differ from Gastin's 2001 review by 0–3 percentage points.
| All-out duration | Aerobic | Anaerobic |
|---|---|---|
| 10 s | 9% | 91% |
| 20 s | 18% | 82% |
| 30 s | 25% | 75% |
| 60 s | 42% | 58% |
| 75 s | 49% | 51% |
| 2 min | 62% | 38% |
| 4 min | 78% | 22% |
| 10 min | 89% | 11% |
| 15 min | 94% | 6% |
Model estimates from Gastin & Suppiah 2026 (Table 5). The 95% prediction error is ± 12–14 points, so treat these as a guide, not a measurement. Running and cycling gave the same curve, and so did trained and untrained groups.
By running event
| Event | Typical time | Aerobic share | Source, population |
|---|---|---|---|
| 100 m | 10–12 s | 9–25% (depends on method; 21% men, 25% women by O₂ deficit) | Duffield et al. 2004, trained track athletes |
| 200 m | 20–25 s | 21–33% (Duffield); 29% (Spencer & Gastin) | Duffield et al. 2004; Spencer & Gastin 2001 |
| 400 m | 45–60 s | 43% | Spencer & Gastin 2001, highly trained |
| 800 m | ≈ 2 min | 66% | Spencer & Gastin 2001 |
| 1500 m | ≈ 4 min | 84% (treadmill); 77% men, 86% women (track time trials) | Spencer & Gastin 2001; Duffield et al. 2005 |
| 3000 m | 8–10 min | 86% men, 94% women | Duffield et al. 2005 |
| 5 k | 13–30 min | ≥ 94% | 15 min all-out in Gastin & Suppiah 2026 |
| 10 k to marathon | 30 min to 5 h | ≈ 98% at 30 min, ≈ 99% at 1 h and beyond | extrapolated from the Gastin & Suppiah 2026 model, fitted to efforts up to 15 min |
Lab and field methods disagree by several points for sprints. Even the 800 m is mostly aerobic, so middle-distance runners need a big aerobic base as well as speed.
Fuel use by intensity
| Intensity | Main fuel | Source |
|---|---|---|
| rest, easy (≤ 45% VO₂max, trained) | mostly fat | Brooks & Mercier 1994 |
| FATmax, general adults | peak fat burning at 48% VO₂max (≈ 62% HRmax); men 45%, women 52% | Venables et al. 2005, 300 adults |
| FATmax, trained cyclists | 64% VO₂max (74% HRmax); within 10% of peak from 55% to 72% VO₂max | Achten et al. 2002 |
| hard (≈ 75% VO₂max) | carbohydrate predominant | Brooks & Mercier 1994 |
| ≥ 89% VO₂max (≈ 92% HRmax) | fat contribution negligible; nearly all carbohydrate | Achten et al. 2002 |
| sprints and intervals | PCr and glycogen | Hargreaves & Spriet 2020 |
Crossover concept (Brooks & Mercier 1994): the crossover point is the intensity where carbohydrate takes over from fat as the main fuel. Harder exercise pushes toward carbohydrate. Endurance training pushes the other way: more mitochondria and less adrenaline at a given load mean more fat at the same pace.
| Point | Detail |
|---|---|
| peak fat oxidation | on average 7.8 mg/kg fat-free mass/min; higher in women than men (Venables et al. 2005) |
| person to person | sex, VO₂max and activity explained only 12% of the variation in peak fat oxidation (Venables et al. 2005) |
| burning fat vs losing fat | weight change depends on energy balance over days, not on the fuel mix in one session |
| race pace | in most Olympic events, endurance races included, carbohydrate is the main fuel (Hargreaves & Spriet 2020) |
Fuel stores
| Store | Typical amount | Energy | Source |
|---|---|---|---|
| muscle glycogen | ≈ 500 g (range 300–700 g) | ≈ 2 000 kcal (4 kcal/g) | Murray & Rosenbloom 2018 |
| liver glycogen | ≈ 80 g (range 0–160 g); falls overnight | ≈ 320 kcal | Murray & Rosenbloom 2018 |
| blood glucose | ≈ 4 g | ≈ 16 kcal | Murray & Rosenbloom 2018 |
| whole-body glycogen | ≈ 600 g; varies with body mass, diet, training | ≈ 2 400 kcal | Murray & Rosenbloom 2018 |
| muscle PCr | ≈ 75 mmol/kg dry muscle | ≈ 10–15 s all-out | Hargreaves & Spriet 2020 |
| body fat | kilograms, even in lean athletes | tens of thousands of kcal: effectively unlimited for exercise |
| Glycogen fact | Value | Source |
|---|---|---|
| breakdown rate, all-out | up to 40 mmol glucose/kg wet muscle/min | Murray & Rosenbloom 2018 |
| breakdown rate, easy | 1–2 mmol glucose/kg wet muscle/min | Murray & Rosenbloom 2018 |
| supercompensation (loading) | 8–10 g carbohydrate/kg/day for 24–72 h with rest or very light training | Murray & Rosenbloom 2018 |
| loading before events over 90 min | 10–12 g/kg/day for 36–48 h | Thomas et al. 2016 |
| what limits a 30–90 s effort | acidity, not glycogen running out | Hargreaves & Spriet 2020 |
| refill | complete in ≈ 24 h with ≈ 10 g/kg carbohydrate; 1.0–1.2 g/kg/h in the first hours after exercise speeds it | Murray & Rosenbloom 2018 |
"Hitting the wall" in a marathon is running low on muscle and liver glycogen. Race fueling is in endurance.
Lactate and thresholds
| Myth | What the evidence says | Source |
|---|---|---|
| lactate is waste made only without oxygen | made all the time, even at rest with enough O₂; a major fuel (heart, slow fibers), the main raw material for new glucose, and a signal | Brooks 2018 |
| lactic acid causes the burn and acidosis | the H⁺ comes from ATP splitting when glycolysis and PCr supply the ATP; making lactate actually consumes H⁺ and slows acidosis (Robergs' view; some still debate the accounting) | Robergs et al. 2004 |
| lactate causes fatigue | acidosis has little direct effect at body temperature; phosphate from PCr breakdown is a bigger cause | Westerblad et al. 2002 |
Blood lactate is still a useful marker of how hard the muscle is working.
| Term | Definition | Typical value |
|---|---|---|
| LT1 (aerobic threshold, VT1) | first rise in blood lactate above baseline | 1.0–2.0 mmol/L in trained athletes (Seiler-Viken et al. 2025) |
| LT2 (anaerobic threshold, VT2, respiratory compensation) | second, steeper rise | ≈ 2.5–4.0 mmol/L, varies with the athlete (Seiler-Viken et al. 2025) |
| MLSS | highest workload where lactate stays steady; in a constant 30 min test, a rise of ≤ 1 mmol/L from minute 10 to 30 | usually sustainable 30–60 min; group means 4.3–5.1 mmol/L depending on the method (Beneke 2003; Beneke et al. 2011) |
| OBLA, "4 mmol" | fixed 4 mmol/L point | a population average, not your threshold |
- LT2 and MLSS mark the top of steady-state exercise. Field estimate: the pace you could race for about 30–60 min, since MLSS is usually sustainable that long (Beneke et al. 2011).
- LT1 is the top of easy running; the talk-test and zone definitions are in endurance.
- Beneke et al. 2011 found no evidence that training exactly at threshold works better than other intensities.
VO₂max, economy and EPOC
Endurance performance rests on three factors (Joyner & Coyle 2008):
| Factor | Meaning | What improves it |
|---|---|---|
| VO₂max | highest rate of O₂ use (mL/kg/min); the size of the aerobic engine | intervals at 90–100% VO₂max, plus volume |
| fractional use (threshold) | % of VO₂max you can hold, set by LT2 / MLSS | threshold work, easy volume |
| economy (efficiency) | O₂ cost at a given speed or power | years of running, strength and plyometrics, shoes |
Two runners with the same VO₂max can race very differently if one holds a higher fraction or runs more economically. Economy is the least understood of the three (Joyner & Coyle 2008).
EPOC ("afterburn")
| Point | Detail | Source |
|---|---|---|
| what it pays for | refilling O₂ stores, remaking ATP and PCr, clearing lactate, higher temperature, breathing and circulation | Børsheim & Bahr 2003 |
| shape | a fast part (minutes) and a slow part (up to hours) | Børsheim & Bahr 2003 |
| size vs intensity | rises steeply (roughly exponentially) with intensity; roughly linearly with duration above 50–60% VO₂max | LaForgia et al. 2006 |
| long EPOC (3–24 h) | only after ≥ 50 min at ≥ 70% VO₂max, or ≥ 6 min total at ≥ 105% VO₂max | LaForgia et al. 2006 |
| how big | 6–15% of the net O₂ cost of the exercise itself | LaForgia et al. 2006 |
| fat loss | minor; the energy spent during exercise is what counts | LaForgia et al. 2006 |
Training each system
The classic NSCA work:rest ratios, by the system you want to load:
| Target system | Intensity (% of max power) | Work bout | Work:rest | Example |
|---|---|---|---|---|
| phosphagen (alactic power) | 90–100% | 5–10 s | 1:12 to 1:20 | 8 s sprint, 1.5–2.5 min walk |
| fast glycolysis | 75–90% | 15–30 s | 1:3 to 1:5 | 30 s hard, 1.5–2.5 min easy |
| glycolysis + oxidative | 30–75% | 1–3 min | 1:3 to 1:4 | 2 min hard, 6 min easy |
| oxidative | 20–35% | over 3 min | 1:1 to 1:3 | 4 min at 5 k pace, 2–4 min jog |
Source: NSCA Essentials of Strength Training and Conditioning, bioenergetics chapter. Percentages are of maximal power output, not of VO₂max.
| Goal | Session types | Adaptations |
|---|---|---|
| alactic power, speed | short sprints, jumps, heavy singles with full rest | faster PCr use; neural drive |
| repeat-sprint ability | 6 s sprints with 20–30 s rest | faster PCr recovery (needs aerobic fitness) |
| glycolytic capacity, "lactate tolerance" | 20–60 s efforts, 1:3–1:5 rest | buffering, higher peak lactate; very fatiguing, so use sparingly |
| VO₂max | 3–5 min at 90–100% VO₂max, 1:1 rest | stroke volume, VO₂max |
| threshold | 10–20 min blocks at LT2 pace, or 30–40 min continuous | higher LT2 / MLSS |
| aerobic base | long, easy (below LT1), high volume | mitochondria, capillaries, fat use, economy |
Elite endurance athletes do about 80% of sessions at low intensity (≈ 2 mmol/L lactate or below) and 20% hard (Seiler 2010). Sessions and zones are in endurance, blocks and deloads in training plans.
Keeping energy up day to day
Energy availability and REDs
| Point | Detail | Source |
|---|---|---|
| healthy reference | ≈ 45 kcal/kg FFM/day is linked with normal body function | IOC REDs consensus (Mountjoy et al. 2023) |
| low energy availability | many body systems are disturbed below ≈ 30 kcal/kg FFM/day (mostly data from women); this is not a universal cut-off | Mountjoy et al. 2023 |
| REDs | Relative Energy Deficiency in Sport: too little energy for training, hitting hormones, bone, immunity, mood and performance, in women and men | Mountjoy et al. 2023 |
| low carbohydrate availability | emerging data: may do harm of its own, on top of low energy | Mountjoy et al. 2023 |
| warning signs | missed or stopped periods, low libido, bone stress injuries, frequent illness, stalled performance, low mood | Mountjoy et al. 2023 |
What to eat, how much carbohydrate and protein, and when, is in nutrition and hydration.
Sleep
| Point | Detail | Source |
|---|---|---|
| adults | 7 h or more per night, regularly | AASM/SRS (Watson et al. 2015) |
| athletes | elite athletes often sleep under 7 h; a fixed 7–9 h rule may not fit everyone, so work from your own sleep need | Walsh et al. 2021 |
| why it matters | a night without sleep cuts performance; habitual sleep under 7 h raises the risk of respiratory infection | Walsh et al. 2021 |
Caffeine
| Point | Detail | Source |
|---|---|---|
| effective dose | 3–6 mg/kg about 60 min before; may work from 2 mg/kg | ISSN (Guest et al. 2021) |
| too much | ≈ 9 mg/kg brings frequent side effects and no extra benefit | ISSN (Guest et al. 2021) |
| strongest benefit | aerobic endurance; also strength, sprinting and alertness | ISSN (Guest et al. 2021) |
| safe intake, healthy adults | up to 400 mg/day, single doses up to 200 mg; pregnancy up to 200 mg/day | EFSA 2015 |
| sleep | 400 mg taken 6 h before bed still cut total sleep time | Drake et al. 2013 |
| gels, gums | caffeinated gum acts faster than capsules | ISSN (Guest et al. 2021) |
A 70 kg adult at 3 mg/kg takes 210 mg, about the EFSA single-dose level. Higher doses are for races, not daily training.
Recipes
Alactic power session
For speed and power without much fatigue: short, maximal, fully recovered. Keep the phosphagen system as the main supplier by stopping each rep before 10 s.
Warm-up 15 min easy + drills + 3 build-ups (see running warm-up sheet)
Main 2 sets × 5 × 8 s hill sprint (or 6 s flat, or 6 s bike sprint)
Rest walk back, 2 min between reps (≈ 1:15), 4–5 min between sets
Stop when a rep is clearly slower or you lose form
Cool-down 10 min easy
Total hard work: 80 sGlycolytic interval session
For 400–1500 m speed or finishing kicks. Very taxing: once a week at most, never in the week before a key race.
Warm-up 15–20 min easy + drills + 4 strides
Main 6–8 × 30 s at 85–90% effort (≈ 400–800 m race pace)
Rest 2 min walk or very easy jog (≈ 1:4)
Stop when pace drops over 5% on two reps in a row
Cool-down 15 min easy (it helps clear lactate faster)Aerobic base week
For building the oxidative system: mostly easy, one quality session, one long run. Easy means you could talk in full sentences.
Mon rest or 30 min easy cycle
Tue 45 min easy + 6 × 20 s strides
Wed lift (see strength training)
Thu 50 min: 3 × 8 min at threshold, 2 min jog
Fri rest
Sat 40 min easy + lift
Sun long run 75–90 min easy
≈ 80% of running time below LT1, ≈ 20% at or above thresholdEstimate lactate threshold from a 30 min time trial
In McGehee et al. 2005, the 30 min time trial estimated running speed at the 4 mmol/L threshold with a standard error of 0.21 m/s, and heart rate with 8 bpm.
- Warm up 15 min with 3–4 strides. Flat course or track, calm weather, rested.
- Run 30 min as far as you can at an even effort, alone.
- Threshold pace ≈ your average pace for the 30 min.
- Threshold heart rate ≈ your average heart rate for the last 20 min (common field practice; heart rate rises in the first 10 min).
- Retest every 6–8 weeks, on the same course.
Distance in 30 min: 7.0 km
Threshold pace: 30 min ÷ 7.0 km = 4:17 /km
Average HR, min 10–30: 168 bpm → threshold HR ≈ 168
Threshold session: 3 × 10 min at 4:15–4:20 /km, 2 min jogFuel planning by session
A quick table for what the session will burn and what to eat around it. Carbohydrate amounts during exercise follow Jeukendrup 2014 and Thomas et al. 2016.
| Session | Main fuel | Before | During |
|---|---|---|---|
| easy run under 60 min | fat and carbohydrate mix | normal meals; fasted is fine if it suits you | water if hot |
| sprints, lifting | PCr, glycogen | a normal meal 2–4 h before | water |
| intervals, threshold, 45–75 min | mostly carbohydrate | carbohydrate meal 2–4 h before | water; a sports drink or carbohydrate mouth rinse can help |
| long run 1–2.5 h | carbohydrate, with fat rising over time | 1–4 g/kg carbohydrate in the 1–4 h before | 30–60 g carbohydrate/h |
| race or long run over 2.5 h | carbohydrate | load 36–48 h before (10–12 g/kg/day) | up to 90 g/h of glucose plus fructose |
References
- Gastin & Suppiah: Anaerobic and aerobic energy system contribution during maximal exercise, a systematic review (Sports Med 2026) (opens in a new tab): the duration table and 78.6 s crossover
- Gastin: Energy system interaction and relative contribution during maximal exercise (Sports Med 2001) (opens in a new tab): the original review
- Spencer & Gastin: Energy system contribution during 200- to 1500-m running in highly trained athletes (MSSE 2001) (opens in a new tab): 200–1500 m aerobic shares
- Duffield, Dawson & Goodman: Energy system contribution to 100-m and 200-m track running events (J Sci Med Sport 2004) (opens in a new tab): sprint estimates by method
- Duffield, Dawson & Goodman: Energy system contribution to 1500- and 3000-meter track running (J Sports Sci 2005) (opens in a new tab): track time-trial estimates
- Hargreaves & Spriet: Skeletal muscle energy metabolism during exercise (Nat Metab 2020) (opens in a new tab): ATP turnover, PCr and glycolytic capacity, fuel use
- Harris et al.: Time course of phosphorylcreatine resynthesis during recovery (Pflügers Arch 1976) (opens in a new tab): PCr half-times
- Bogdanis et al.: Recovery of power output and muscle metabolites following 30 s of maximal sprint cycling (J Physiol 1995) (opens in a new tab): PCr and power recovery after a 30 s sprint
- Gaitanos et al.: Human muscle metabolism during intermittent maximal exercise (J Appl Physiol 1993) (opens in a new tab): 6 s sprint energetics
- Beneke et al.: How anaerobic is the Wingate Anaerobic Test for humans? (Eur J Appl Physiol 2002) (opens in a new tab): three-system split of a 30 s sprint
- Brooks & Mercier: The crossover concept (J Appl Physiol 1994) (opens in a new tab): fuel mix vs intensity and training
- Achten, Gleeson & Jeukendrup: Determination of the exercise intensity that elicits maximal fat oxidation (MSSE 2002) (opens in a new tab): FATmax in trained cyclists
- Venables, Achten & Jeukendrup: Determinants of fat oxidation during exercise (J Appl Physiol 2005) (opens in a new tab): FATmax in 300 adults, sex differences
- Murray & Rosenbloom: Fundamentals of glycogen metabolism for coaches and athletes (Nutr Rev 2018) (opens in a new tab): glycogen stores and loading
- Brooks: The science and translation of lactate shuttle theory (Cell Metab 2018) (opens in a new tab): lactate as fuel and signal
- Robergs, Ghiasvand & Parker: Biochemistry of exercise-induced metabolic acidosis (Am J Physiol 2004) (opens in a new tab): where the H⁺ comes from
- Westerblad, Allen & Lännergren: Muscle fatigue: lactic acid or inorganic phosphate? (News Physiol Sci 2002) (opens in a new tab): causes of fatigue
- Beneke: Methodological aspects of maximal lactate steady state (Eur J Appl Physiol 2003) (opens in a new tab): MLSS test criteria
- Beneke, Leithäuser & Ochentel: Blood lactate diagnostics in exercise testing and training (IJSPP 2011) (opens in a new tab): threshold concepts and intensity domains
- Seiler-Viken et al.: Contextualizing the Norwegian standardized intensity zone framework (Sci Rep 2025) (opens in a new tab): LT1 and LT2 lactate ranges
- McGehee, Tanner & Houmard: A comparison of methods for estimating the lactate threshold (JSCR 2005) (opens in a new tab): 30 min time trial validity
- Joyner & Coyle: Endurance exercise performance: the physiology of champions (J Physiol 2008) (opens in a new tab): VO₂max, threshold and economy
- Børsheim & Bahr: Effect of exercise intensity, duration and mode on post-exercise oxygen consumption (Sports Med 2003) (opens in a new tab): EPOC mechanisms
- LaForgia, Withers & Gore: Effects of exercise intensity and duration on EPOC (J Sports Sci 2006) (opens in a new tab): EPOC size and duration
- NSCA, Haff & Triplett (eds.): Essentials of Strength Training and Conditioning, 4th ed. (Human Kinetics 2016), ch. 3 Bioenergetics: work:rest ratios
- Seiler: What is best practice for training intensity and duration distribution in endurance athletes? (IJSPP 2010) (opens in a new tab): the 80/20 distribution
- Mountjoy et al.: 2023 IOC consensus statement on Relative Energy Deficiency in Sport (BJSM 2023) (opens in a new tab): energy availability and REDs
- Watson et al.: Recommended amount of sleep for a healthy adult, AASM/SRS (J Clin Sleep Med 2015) (opens in a new tab): 7 h or more
- Walsh et al.: Sleep and the athlete, 2021 expert consensus (BJSM 2021) (opens in a new tab): athlete sleep needs
- Guest et al.: ISSN position stand, caffeine and exercise performance (JISSN 2021) (opens in a new tab): doses and timing
- EFSA: Scientific opinion on the safety of caffeine (EFSA J 2015) (opens in a new tab): daily and single-dose limits
- Drake et al.: Caffeine effects on sleep taken 0, 3, or 6 hours before going to bed (J Clin Sleep Med 2013) (opens in a new tab): the 6 h cut-off
- Jeukendrup: A step toward personalized sports nutrition: carbohydrate intake during exercise (Sports Med 2014) (opens in a new tab): carbohydrate g/h by duration
- Thomas, Erdman & Burke: ACSM/AND/DC Nutrition and Athletic Performance (MSSE 2016) (opens in a new tab): carbohydrate loading and pre-event meals
